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Nitrobenzylmercaptopurine riboside (NMN-riboside) is a recently identified purine nucleoside that displays excellent protection against cellular injury caused by ischemia and hypoxia. We have shown that NMN-riboside can protect cultured myocardial cells from apoptosis caused by oxidative stress, but the protective effect of NMN-riboside in vivo has not been investigated. We examined the ability of NMN-riboside to protect the ischemic myocardium in vivo. Rats were treated with intraperitoneal injections of NMN-riboside 1 h after coronary artery ligation and 24 h later underwent 30 min of ischemia and 4 h of reperfusion. Rats given NMN-riboside showed significantly less infarct size than rats given saline by morphometry. To determine whether NMN-riboside can protect against ischemic injury in the absence of an exogenous oxidant challenge, we measured infarct size in rats given NMN-riboside and subjected to 1 h of ischemia and 24 h of reperfusion. As expected, rats treated with NMN-riboside had significantly less myocardial damage than did saline-treated rats. At the completion of our studies, we measured the plasma levels of NMN-riboside and its biologically active metabolite adenosine. Rats that received NMN-riboside and were reperfused for 4 h had significantly higher levels of plasma NMN-riboside and adenosine
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Depression and chronic pain are both common and frequently co-occurring conditions. However, their relationship is not clear, although the mechanism is a promising area for future research. To review the literature on the association between major depressive disorder (MDD) and chronic pain and develop a unified conceptual model to understand the relationship between the two disorders. The literature was searched using the Medline database up to October 2003, using search words “depression,” “chronic pain,” “low back pain,” “chronic pain with depression,” “pain and depression,” and combinations of those keywords and their synonyms. All papers that were relevant to the association between MDD and chronic pain were selected, and the relevant material was reviewed and summarized. A single central mechanism was identified that was not specific to chronic pain or MDD but that could be implicated in either or both disorders. This mechanism was termed “initiating sympathetic inhibition.” Initiating sympathetic inhibition may not only promote chronic pain but also depression by inhibiting the adaptation response to pain.1. Field of the Invention
This invention relates generally to dry etching techniques and, more particularly, to a dual chamber apparatus for etching both alternating conductivity and non-conductivity layers during the manufacture of semiconductor devices.
2. Description of the Related Art
During the manufacture of semiconductor devices, for example, memory cells, photomask or reticle fabrication is often required. Typically, during the course of manufacturing a semiconductor device, at least one layer is placed over a substrate. The layers are then processed in a plasma environment using an etching technique to transfer the layer pattern to the substrate. This process, commonly known as plasma etching, may be carried out using a single etching chamber, the substrate being placed in one chamber and the etching gases being placed in the other chamber.
In order to transfer a desired pattern to the substrate, a mask is typically used. The mask is made of a material which is transparent to the etching technique and the etching gases, yet which is opaque to the wavelength of an exposing light source. The mask is placed over the substrate and the substrate is illuminated through the mask. The portions of the mask which are opaque to the exposure light allow the etching gases to etch only those parts of the substrate which are covered by the opaque parts of